Today endocrinologists are discussing the best methods of treatment in hypo-and hyperthyroidism, according to the specific needs of hormone replacement. Most physicians base their indications on the use of levo-thyroxine is actually the thyroid hormone T4 which gradually will release T4 and T3 both substances. the oldest form of administration of triiodothyronine stronger, but shorter-acting (T3) is slowly being replaced by more biological T4.

A new branch of modern medicine, alternative medicine does not believe in the beneficial effects of hormone use is only T4. Human thyroid gland is known to secrete three different types of hormones, T3, T4 and DIT called diiodothyrosine. it has been demonstrated clinically that patients who suffer from hypothyroidism, with symptoms that show a low improvement after the treatment with T4, have shown a significant change in thyroid extracts well when added to medication.

In a recent study in rats thyroidectomised have approved the final date ideas in alternative medicine specialists. Rats treated with T4 alone showed normal levels of T4 in the tissues and plasma, but lower levels of T3 in the tissue itself, while the amount of TSH thyroid stimulating hormone remained elevated. after these findings the scientists decided to administer 0.9 mcg of T4 and T3 0.15 mcg per day and per 100 g of body weight. the findings were beneficial as both T3 and T4 levels returned to normal and the TSH level.

This study has not investigated the role of DIT hormone in thyroid function and the need for administration on thyroid dysfunction is still unknown. DIT role has however been found in the treatment of fibrocystic breast disease and the treatment of thyreotoxicosis.

The study presented has been able to feel their goals to show that nature can be mimicked by administration of hormones such as assembly secreted by the gland under normal circumstances. the body’s own glandular function can be replaced artificially supplementing the main idea is actually that we imitate nature, but can not improve it and make it more perfect than it already is.

The most interesting studies are finding the best method of administration of replacement hormones. T3 is usually more rapid oral absorption and a combination of T3 and T4, as temporary Euthroid has raised the level of T3 leads to stress to the nervous system. First conclusion is that exact same thyroid hormones given orally can not produce the same effect as secreted by the thyroid gland and released into the bloodstream.

mood swings

weight loss?

Some symptoms can be losing 15% or more of body weight. A person who has Anorexia may abuse laxatives, diuretics, and enemas. S/he may also self induce vomiting. for women, menstrual periods may stop happening. there are also symptoms due to malnutrition which are: constipation, digestive discomfort, bloating, dehydration, muscle cramps, and tremors; downy body hair on the face, back, or arms; dull, brittle, thinning hair; flattened breasts, cracked or dry skin, icy hands and/or feet, irregular heartbeat, depression and/or anxiety. Anorexia also causes low levels of estrogen and thyroid hormone, and increased levels of cortisol.

*Suspect a problem if you:
-Have lost at least 15% percent of your weight without a physical illness and still think of yourself as fat.
-Have not had your period in three months in a row.
-Fear being overweight and losing control of your diet.
-Feel that staying hungry is the only way to keep your weight under control.

Accepted wisdom that there is no physiologically important transport of thyroid hormones during development and that hormones would not be necessary to regulate fetal growth of the nervous system is likely to have contributed to a common lack of understanding and acceptance of the results of many epidemiological and clinical studies. A credible reason could not be proposed for the association between early maternal hypothyroxinemia result of the reduction of infant neurological development. It is difficult to solve the very serious and often permanent, injuries, neurological cretins (with normal thyroid function when iodine supply is complete), with the successful prevention of severe nervous system injuries early postnatal management of infants congenital hypothyroidism, counting children without the thyroid gland.

The lack of defensive actions maternal triiodothyronine (T3) compared to thyroxine (T4), shown in iodine-deficient regions deep, show many problems unresolved. Original held frequently purchased over the past 15 years in experimental animals and man, now evidently point to a function of the location of the mother of thyroid hormone in the mature nervous system for life fetal and suggest reasonable detail the above issues. Regardless of these new results and even with the increased recognition of a significant role in maternal T4 in the impediment of a serious nervous system damage in cases of congenital hypothyroidism, is still said that if thyroid hormone is needed throughout the 1st quarter is less certain, otherwise must be supplied by the mother, because nothing is released by the product until the second trimester.

Since this problem is fundamental to understanding the possible significance of thyroxine in the first quarter free (FT4) in fetal maturation of the nervous system, we must pay attention to the data currently available for experimental animals and humans. both T4 and T3 are available to the fetus early with very low amounts found in amounts that circulate in the mother are significantly low. As soon as this happens, changes in prenatal and postnatal development may be revealed. Nuclear receptors for thyroid hormones (TR) are also in the early stages of fetal development of the nervous system to some extent occupied by T3, in some animal models, with increasing levels at all times extremely vigorous birth of neurons in the cortex.

Several genes in the nervous system responds to thyroid hormone deficiency has been recognized frequently in the post-natal period, through a time of expansion of the nervous system corresponding to the last period of pregnancy and early postnatal period humans.